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ostreatus, whereas only cytosolic catalase was overexpressed in P. cornucopia, suggesting their different Cd detoxification pathways. Also, the mitogen-activated protein kinase signaling pathway involved in Cd resistance in both species instead of glutathione metabolism, although more active in P. ostreatus. These findings provided new insight into the molecular mechanism of mycoremediation and accumulator screening.In this work, we have reported on the facile synthesis of white light-emitting carbon quantum dots (CQD) from corncob by hydrothermal method. This CQD has a broad emission from 380 nm to 650 nm with high photoluminescence intensity even after three months of shelf-life and stable at variable pH conditions. The presence of Si and N impurities in the biomass gives a greater advantage in producing white light emission with high quantum yield (54%) and enhanced lifetime at ambient conditions. The CQD is highly sensitive towards DNA, paracetamol, Pb2+, Cu2+, Fe3+, and Cr3+ fluorescence sensing and signifies its application as a multi-modal fluorescence sensor. The results of optical sensitivity calculated from the linear range of 1-10 ng/mL, 0.10-0.30 mg/mL, 2.5446 ng/mL, 0.0694 mg/mL, 0.3103-1.5515 μM/mL, 0.4299-4.7293 μM/mL, 1.3010 μM/mL and 0.05-2.5 μM/mL. The limit of detection is 2.5446 ng/mL, 0.0694 mg/mL, 0.8641 μM/mL, 1.2454 μM/mL, 1.3010 μM/m, 0.8550 μM/mL and 2.8562 μM/mL, respectively. And also, the relative standard deviation values of 2.30%, 4.46%, 1.79%, 1.84%, 0.26%, 1.23% and 0.35% are evidences its possibility of development towards potential optical sensor applications. Flexible white light-emitting sheets were fabricated from the CQD, illuminates uniform brightness, and has good color reproducibility and higher stability under various UV light excitation.A novel K-resistant Fe3+ and Zr4+ co-doped CeTiOx catalyst was first prepared by co-precipitation method for the ammonia-selective catalytic reduction (NH3-SCR) of NOx. On the premise of retaining the outstanding catalytic activity of CeTiOx catalyst, Fe3+ and Zr4+ co-doping efficiently improves its K-resistance with superior NOx conversion up to 84% after K-poisoning. Specially, the grain growth during the second calcination after K poisoning is successfully inhibited by Fe3+ and Zr4+ co-doping. Consequently, the large specific surface area with increased acid sites and efficiently retained reducibility over K-poisoned FeZrCeTiOx catalyst are realized, which prompt NH3 activation and NO oxidation, further benefit NH3-SCR. buy Olcegepant Besides, NH3-SCR reaction over CeTiOx and FeZrCeTiOx catalysts follows a possible L-H mechanism, and K-poisoning makes no change to it. Finally, a reasonable anti-K poisoning mechanism of FeZrCeTiOx catalyst is proposed the excellent K-resistance is attributed to part of Fe and Zr are sacrificed to form Fe-O-K and Zr-O-K species protecting the active site Ce-O-Ti from K-poisoning, as well as the additional reducibility and surface acidity brought from Fe-O species with Zr prompting its uniform distribution.Microplastics (MPs) are emerging pollutants that act as a carrier of toxic pollutants, release toxic substances, and aggregate in biota. The adsorption behavior of MPs has recently become a research hot spot. The objective of this study was to summarize the main mechanisms by which MPs adsorb organic pollutants, introduce some mathematical models commonly used to study the adsorption behavior of MPs, and discuss the factors affecting the adsorption capacity from three perspectives, i.e., the properties of MPs and organic pollutants, and environmental factors. Adsorption kinetics and isothermal adsorption models are commonly used to study the adsorption of organic pollutants on MPs. We observed that hydrophobic interaction is the most common mechanism by which MPs adsorb organic pollutants, and also reportedly controls the portion of organic pollutants. Additionally, electrostatic interaction and other non-covalent forces, such as hydrogen bonds, halogen bonds, and π-π interactions, are also mechanisms of organic pollutant adsorption on MPs. The particle size, specific surface area, aging degree, crystallinity, and polarity of MPs, and organic pollutant properties (hydrophobicity and dissociated forms) are key factors affecting adsorption capacity. Changes in the pH, temperature, and ionic strength also affect the adsorption capacity. Current research on the adsorption behavior of MPs has mainly been conducted in laboratories, and in-depth studies on the adsorption mechanism and influencing factors are limited. Therefore, studies on the adsorption behavior of MPs in the environment are required, and this study will contribute to a better understanding of this topic.1,4-Dioxane (dioxane), an emerging groundwater contaminant, is frequently detected in landfill leachates with its structural analog, tetrahydrofuran (THF). Along with undesirable leakage of landfill leachates, dioxane and THF inevitably percolate into groundwater leading to a broader region of contamination. Cometabolic bioremediation is an effective approach to manage commingled THF and dioxane pollution. In this study, a newly isolated bacterium Arthrobacter sp. WN18 is able to co-oxidize dioxane with THF as the primary substrate. Meanwhile, the THF-induced thmADBC gene cluster was responsible for the dioxane degradation rate indicating THF monooxygenase is the essential enzyme that initializing α-hydroxylation of THF and dioxane. Further, γ-butyrolactone and HEAA were characterized as the key metabolites of THF and dioxane, respectively. In addition, WN18 can tolerate the inhibition of trichloroethylene (5.0 mg/L) as a representative of co-existing leachate constituent, and sustain its activity at various pH (5-11), temperatures (15-42 °C), and salinities (up to 4%, as NaCl wt). Like other Arthrobacter species, WN18 also exhibited the capability of fixing nitrogen. All this evidence indicates the feasibility and advantage of WN18 as a thmADBC-catalyzed inoculator to bioremediate co-contamination of THF and dioxane.Ig-GAD2, an antigen-specific immune modulator, requires bone marrow (BM) cell transfer in order to restore beta (β)-cell formation and induce recovery from established type 1 diabetes (T1D). The BM cells provide endothelial precursor cells (EPCs) that give rise to islet resident endothelial cells (ECs). This study shows that, during development of T1D, the immune attack causes collateral damage to the islet vascular network. The EPC-derived ECs repair and restore islet blood vessel integrity. In addition, β-cell genetic tracing indicates that the newly formed β-cells originate from residual β-cells that escaped the immune attack and, unexpectedly, from β-cell precursors. This indicates that the rejuvenated islet microenvironment invigorates formation of new β-cells not only from residual β-cells but also from precursor cells. This is twofold significant from the perspective of precursor cells as a safe reserve for restoration of β-cell mass and its promise for therapy of T1D long after diagnosis.