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5%) and CI100-200 (-8.9%). In conclusion, older men demonstrated greater variability for maximal torque output, but lesser for rapid torque.

The risk of exercise-induced endotoxemia is increased in the heat and is primarily attributable to changes in gut permeability resulting in the translocation of lipopolysaccharides (LPS) into the circulation. The purpose of this study was to quantify the acute changes in gut permeability and LPS translocation during submaximal continuous and high-intensity interval exercise under heat stress.

A total of 12 well-trained male runners (age 37 [7]y, maximal oxygen uptake [VO2max] 61.0 [6.8]mL·min-1·kg-1) undertook 2 treadmill runs of 2 × 15-minutes at 60% and 75% VO2max and up to 8 × 1-minutes at 95% VO2max in HOT (34°C, 68% relative humidity) and COOL (18°C, 57% relative humidity) conditions. SR-18292 price Venous blood samples were collected at the baseline, following each running intensity, and 1 hour postexercise. Blood samples were analyzed for markers of intestinal permeability (LPS, LPS binding protein, and intestinal fatty acid-binding protein).

The increase in LPS binding protein following each exercise intensity in the HOT condition was 4% (5.3μg·mL-1, 2.4-8.4; mean, 95% confidence interval, P < .001), 32% (4.6μg·mL-1, 1.8-7.4; P = .002), and 30% (3.0μg·mL-1, 0.03-5.9; P = .047) greater than in the COOL condition. LPS was 69% higher than baseline following running at 75% VO2max in the HOT condition (0.2 endotoxin units·mL-1, 0.1-0.4; P = .011). Intestinal fatty acid-binding protein increased 43% (2.1ng·mL-1, 0.1-4.2; P = .04) 1 hour postexercise in HOT compared with the COOL condition.

Small increases in LPS concentration during exercise in the heat and subsequent increases in intestinal fatty acid-binding protein and LPS binding protein indicate a capacity to tolerate acute, transient intestinal disturbance in well-trained endurance runners.

Small increases in LPS concentration during exercise in the heat and subsequent increases in intestinal fatty acid-binding protein and LPS binding protein indicate a capacity to tolerate acute, transient intestinal disturbance in well-trained endurance runners.

Regular physical exercise can attenuate age-related cognitive decline. This study aimed to investigate the effect of a physical exercise multicomponent training based on exergames on cognitive functioning (CF) in older adults.

This randomized controlled trial included older adults aged 61-78. Participants were randomly allocated to an intervention group (IG; n = 15) or active control group (CG; n = 16). The IG was exposed to a combined training with traditional exercise and exergaming, twice a week over a period of 12 weeks. The CG performed only traditional sessions. CF was assessed by the Cognitive Telephone Screening Instrument. The time points for assessment were at zero (pretest), 12 (posttest), and 17 weeks (follow-up).

Active CG and IG increased from pretest to posttest in short-term memory (STM), long-term memory (LTM), and Cognitive Telephone Screening Instrument total score 1.98 > Z < 3.00, ps < .005, with moderately large positive effects (.36 > r < .54). A significant increasend Implications The integration of exergaming in a multicomponent functional fitness exercise might have the potential to maintain and improve CF (in particular, STM and LTM) in older adults.The authors compared the effects of bodyweight resistance training at moderate- or high-speed conditions on muscle power, velocity of movement, and functional performance in older females. In a randomized, single-blinded noncontrolled trial, participants completed 12 weeks (three sessions/week) of bodyweight resistance training at high (n = 14; age = 70.6 ± 4.3 years) or moderate (n = 12; age = 72.8 ± 4.2 years) speeds. Data were analyzed with an analysis of variance (Group × Time) with α level set at .2). The authors conclude that high-speed bodyweight resistance training is an effective and economically practical strategy to improve the functional capacity of older women relevant to daily life activities.

Chronic ankle instability is documented to be followed by a recurrence of giving away episodes due to impairments in mechanical support. The application of ankle Kinesiotaping (KT) as a therapeutic intervention has been increasingly raised among athletes and physiotherapists.

This study aimed to investigate the impacts of ankle KT on the lower-limb kinematics, kinetics, dynamic balance, and muscle activity of college athletes with chronic ankle instability.

A crossover study design.

Twenty-eight college athletes with chronic ankle sprain (11 females and 17 males, 23.46 [2.65]y, 175.36 [11.49]cm, 70.12 [14.11]kg) participated in this study.

The participants executed 3 single-leg drop landings under nontaped and ankle Kinesio-taped conditions. Ankle, knee, and hip kinematics, kinetics, and dynamic balance status and the lateral gastrocnemius, medial gastrocnemius, tibialis anterior, and peroneus longus muscle activity were recorded and analyzed.

The application of ankle KT decreased ankle joint rangitable supportive means for acute usage in athletes with chronic ankle instability.A popular posture for using wireless technology is reclined sitting, with the trunk rotated posteriorly to the hips. This position decreases the head's gravitational moment; however, the head angle relative to the trunk is similar to that of upright sitting when using a tablet in the lap. This study compared cervical extensor musculotendon length changes from neutral among 3 common sitting postures and maximum neck flexion while using a tablet. Twenty-one participants had radiographs taken in neutral, full-flexion, and upright, semireclined, and reclined postures with a tablet in their lap. A biomechanical model was used to calculate subject-specific normalized musculotendon lengths for 27 cervical musculotendon segments. The lower cervical spine was more flexed during reclined sitting, but the skull was more flexed during upright sitting. Normalized musculotendon length increased in the reclined compared with an upright sitting position for the C4-C6/7 (deep) and C2-C6/7 (superficial) multifidi, semispinalis cervicis (C2-C7), and splenius capitis (Skull-C7).

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