Konradsenmonahan8919

001). At the end, 50% of the control animals had paraplegia compared with 0% of paraplegia in the priming group. The mean regional histopathologic score differed between the priming group and the control group (P=.02). The priming group had higher motor-evoked potentials during the operation at separate time points. The lactate levels were lower in the priming group compared with the control group (P

=.001, P

=.18).

Acute priming protects the spinal cord from ischemic injury in an experimental aortic crossclamp model.

Acute priming protects the spinal cord from ischemic injury in an experimental aortic crossclamp model.

Postoperative atrial fibrillation (POAF) is a common complication after coronary artery bypass grafting (CABG). Currently, there is no reliable way to determine preoperatively which patients will develop POAF following CABG. The aim of this study was to determine whether preoperative left atrial (LA) strain analysis might identify patients destined to develop POAF following CABG.

From 2016 to 2018, 211 patients who had a preoperative left ventricular ejection fraction >50% and adequate preoperative, predischarge, and follow-up echo images for interpretation underwent isolated CABG surgery. Postoperatively, patients had continuous rhythm monitoring until hospital discharge. Retrospective speckle-tracking analysis of preoperative echocardiograms was performed to calculate preoperative left ventricular global longitudinal strain and LA compliance and contraction strains in 92 matched patients. Multivariate logistic regression and Cox proportional hazards models were used to determine the predictors of POAly LA-fractional area change, LA-emptying fraction, and LA-reservoir strain, taken jointly, are more specific and sensitive than other preoperative parameters in identifying patients who will develop POAF following CABG. The ability to identify patients preoperatively who are destined to develop POAF following CABG provides a basis for limiting POAF prophylactic therapy to only those patients undergoing CABG who are most likely to benefit from it rather than to all patients undergoing CABG.

The impact of coronavirus disease 2019 (COVID-19) on the postoperative course of patients after cardiac surgery is unknown. We experienced a major severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) outbreak in our cardiac surgery unit, with several patients who tested positive early after surgery. Here we describe the characteristics, postoperative course, and laboratory findings of these patients, along with the fate of the health care workers. We also discuss how we reorganize and reallocate hospital resources to resume the surgical activity without further positive patients.

After diagnosis of the first symptomatic patient, surgery was suspended. Nasopharyngeal swabs were performed in all patients and health care workers. Patients who were positive for SARS-CoV-2 were isolated and monitored throughout the in-hospital stay and followed up after discharged until death or clinical recovery.

Twenty patients were found to be positive for SARS-CoV-2 sometime after cardiac surgery (mean age 69±10.oreal circulation may mask the typical COVID-19 laboratory findings, making the diagnosis more difficult. A strict reorganization of the hospital resources is necessary to safely resume the cardiac surgical activity.

To characterize changes in Fontan conduit size over time and determine if cross-sectional area (CSA) affects cardiac output, pulmonary artery growth, and exercise capacity.

We conducted a retrospective cross-sectional study of patients with Fontan physiology who underwent cardiac magnetic resonance imaging or cardiac catheterization between January 2013 and October 2019. We collected Fontan and pulmonary artery measurements, hemodynamic data, and cardiopulmonary exercise test data. We identified 158 patients with an extracardiac Fontan. We measured minimum and mean Fontan conduit CSA and assessed whether these correlated with Nakata index, cardiac index, or exercise capacity.

Minimum Fontan CSA decreased by a median of 33% (24%, 40%) during a mean follow-up of 9.6years. Median percentage decrease in Fontan CSA did notdiffer among 16-, 18-, and 20-mm conduits (P=.29). There was a significant decrease in the minimum Fontan CSA (33% [25%, 41%]) starting less than 1-year post-Fontan. Median Nakata index was 177.6mm

/m

(149.1, 210.8) and was not associated with Fontan CSA/BSA (ρ=0.09, P=.29). Fontan CSA/BSA was not associated with cardiac index (ρ=-0.003, P=.97). A larger Fontan CSA/BSA had a modest correlation with % predicted oxygen consumption (ρ=0.31, P=.013).

Fontan conduit CSA decreases as early as 6months post-Fontan. The minimum Fontan CSA/BSA was not associated with cardiac index or pulmonary artery size but did correlate with % predicted peak oxygen consumption.

Fontan conduit CSA decreases as early as 6 months post-Fontan. The minimum Fontan CSA/BSA was not associated with cardiac index or pulmonary artery size but did correlate with % predicted peak oxygen consumption.The protective performance of graphene/polymer composite coatings largely depends on the interface design in resin matrix. Herein, we report the synthesis of bio-based cardanol epoxy modified graphene oxide (GODN) nanomaterial and its application in epoxy coatings for the achievement of fine interface toward high performance anticorrosion composite coatings. The chemical composition of prepared GODN nanomaterial was investigated by FTIR, Raman and XPS spectra, respectively. The presence of cardanol epoxy attached on GO surface promotes the formation of chemical bonds between GO and epoxy resin, providing strong interfacial interaction and enhanced adhesion. CH-223191 datasheet Electrochemical results revealed that the GODN1%/EP composite coating exhibits high impedance (4.38 × 108 Ω cm2) even after 45 days immersion. Compared with pure EP coating, the localized corrosion reaction of GODN1%/EP coating can be inhibited under defected interface. The enhanced protective performance of GODN/EP composite coating was attributed to two aspects (1) the impermeable GO greatly suppressed the penetration of aggressive ions and (2) the attached cardanol epoxy chains effectively improved the interfacial interaction and thus inhibited the crack propagation.